This study uncovers that activation of endogenous retroviruses affects prion-like spreading of proteopathic seeds. It supports that endogenous retroviruses play a role in protein misfolding diseases and suggest that antiviral drugs could represent promising candidates for inhibiting protein aggregate spreading.
https://www.nature.com/articles/s41467-023-40632-z (open access)
2 Comments
solrize@lemmy.world · 2 pts · 2y
Interesting. Does this mean Alzheimer's might come from an ERV?
Bebo@literature.cafe · 2 pts · 2y
At least the ERVs seem to play a role in the progression of the disease.